KVV E-Modules - Pathophysiology of Hypertension

Quadrant -I (Video)

Video on Pathophysiology of hypertension

Quadrant - II (Study Material)

1. Notes on Pathophysiology of Hypertension

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Quadrant - III (Assignments)

1. Assignment on Pathophysiology of Hypertension

A 52-year-old man with no known disease history is diagnosed with Stage I hypertension. He has high dietary sodium intake, poor physical activity, and chronic stress. Lab reports show elevated plasma renin activity and borderline kidney function. Questions: 1. Based on this case, what mechanisms are likely contributing to his hypertension? 2. Suggest one lifestyle and one pharmacological intervention with justification.

Quadrant - IV (MCQs)

Primary hypertension accounts for approximately:

A: 10% of cases B: 25% of cases C: 90–95% of cases D: 5% of cases

Secondary hypertension is caused by:

A: Unknown factors B: Lifestyle only C: Genetic mutation D: Identifiable underlying conditions

Which hormone system is most involved in long-term BP regulation?

A: Adrenal system B: Renin-Angiotensin-Aldosterone System (RAAS) C: Pituitary axis D: Pineal gland

Baroreceptors are primarily involved in:

A: Long-term BP regulation B: Short-term BP regulation C: Metabolism D: Renal filtration

Which of the following is not a typical cause of secondary hypertension?

A: Renal artery stenosis B: Adrenal tumors C: Lifestyle stress D: Drug-induced effects

Angiotensin II causes:

A: Vasodilation B: Vasoconstriction C: Decreased blood volume D: Bradycardia

Aldosterone increases blood pressure by:

A: Increasing water and sodium excretion B: Blocking renin release C: Increasing sodium and water retention D: Causing vasodilation

Endothelial dysfunction leads to:

A: Increased nitric oxide release B: Improved vasodilation C: Reduced vascular resistance D: Impaired vasodilation and arterial stiffness

Antidiuretic hormone (ADH) affects BP by:

A: Promoting fluid loss B: Reducing renal blood flow C: Promoting water retention D: Blocking sodium channels

A consequence of reduced nitric oxide production in blood vessels is:

A: Vasodilation B: Hypertension C: Bradycardia D: Decreased cardiac output

Validation Certificates

1. E content module certificate (Validation Part I)

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2. E - Content module certificate (Validation Part II)

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3. Checking report

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